Smith Seminars Online Mechanisms of COPD Test

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If for some reason you do not pass the test with 70% correct you will be notified by email to retake the test. Your order confirmation number will stay valid until you pass the test.

 

 Part 1 - Your Information

 

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 Part 2 - The Test

 

1) Chronic Obstructive Pulmonary Disease or COPD:

A) Is highly reversible

B) Is characterized by slowly progressive development of airflow limitation

C) Is identical to asthma

D) None of the above

2) Mechanisms of airflow limitation of the small airways include:

A) Narrowing of small airways in patients with COPD

B) An increase in the thickness of small airways with increased formation of lymphoid follicles

C) Deposition of collagen in the outer airway wall that may restrict airway opening

D) All the above

3) An increase in the inflammatory cell types implicated in COPD include:

A) Macrophages

B) T- and B-lymphocytes

C) Neutrophils

D) All the above

4) Neutrophils:

A) Have the capacity to induce tissue damage through the release of serine proteases and oxidants

B) Recruited to the airways of COPD patients are deactivated granule proteins in the sputum

C) A & B

D) None of the above

5) Macrophages:

A) May be activated by cigarette smoke extract

B) Secrete more inflammatory proteins

C) Have a long survival time so this is difficult to measure directly
D) All the above

6) The airways and lungs contain a rich network of dendritic cells:
A) That are localized near the surface
B) Are ideally located to signal the entry of foreign substances that are inhaled
C) That can activate a variety of other inflammatory and immune cells
D) All the above

7) Airway epithelial cells:
A) Secrete dextrose and other catabolic proteins with antimicrobial effects
B) Secrete antioxidants and transport immunoglobulin Z
C) Are important in defense of the airways
D) All the above

8) The increased oxidative stress in the airways of COPD patient:
A) May play an important pathophysiological role in the disease by amplifying the inflammatory response in COPD
B) May impair the function of proteases and decelerate the breakdown of elastin in lung parenchyma
C) A & B
D) None of the above

9) High doses of corticosteroids:
A) Cause an increase in histone deacetylase activity in macrophages
B) Fail to reduce cytokine and chemokines that should be suppressed by corticosteroid treatment
C) A & B
D) None of the above

10) Persistence of certain bacteria in the lower respiratory tract of COPD patients:
A) May be due to localization to protected sites
B) Is always susceptible to antibiotics or antibody-mediated defense mechanisms when they are located between airway epithelial cells
C) Is associated with decreased concentrations of inflammatory mediators in COPD patients
D) None of the above

 

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Smith Seminars

PO Box 516 · Paradise · TX · 76073 

Phone 866-857-2211 · Fax 972-759-9197
Email debra@smithseminars.com

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